Abstract
Extracellular-regulated kinase 3 (ERK3) - an atypical mitogen-activated protein kinase (MAPK), coded by the MAPK6 gene - is overexpressed in breast cancer (BC), strongly correlating with poor patient survival. However, the mechanisms by which ERK3 contributes to BC remain unknown. Therefore, this study aims to elucidate the role of ERK3 in breast cancer progression, especially in aggressive triple-negative BC subtype (TNBC).
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