Abstract
Rats maintained on tritiated 1,25-dihydroxyvitamin D 3 as their sole source of vitamin D and placed on diets differing in calcium content had similar intestinal levels of tritiated 1,25-dihydroxyvitamin D 3. Since 1,25-dihydroxyvitamin D 3 administration eliminated adaptation of intestinal calcium transport, it appears that increased production of 1,25-dihydroxyritamin D 3 is responsible for the stimulation of calcium transport by low dietary calcium. When maintained on tritiated 1,25-dihydroxyvitamin D 3, rats fed a low-phosphorus diet had somewhat higher levels of tritiated 1,25-dihydroxyvitamin D 3 in the duodenum and plasma than rats on a normal-phosphorus diet. In addition to stimulating 1,25-dihydroxyvitamin D 3 synthesis, low dietary phosphorus may increase the accumulation of 1,25-dihydroxyvitamin D 3 in both intestine and plasma.
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