Abstract
Neonates are known to regulate CO by changing HR. Little is known on changes of stroke volume in sick infants. We measured CO,SV,HR and mean arterial blood pressure (BP) by Dopplersonography in 40 sick infants before(A,B) and after (Aτ, Bτ) therapy. (A):low CO due to respiratory problems(n=15).(B): high CO due to PDA(n=25). (C) and (D): Normal infants(n=40) matched for GA(control). In (A) SV was low and not compensated by a high HR. BP was normal. The rise of CO after treatment(Atau;) was due to a rise in SV. In (B) SV was high and normalized after ductus closure (Btau;), BP was low and increased. HR did not change. Conclusion: Neonates with low CO and low preload do not compensate low SV by increasing the HR. Preterm infants can increase their SV above normal levels if preload is high and afterload is low. CO in sick neonates is not only regulated by HR but also by SV. Results (mean ± SD):
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