Abstract
Aging is accompanied by the decline of biological and physical functions in living organisms. It is widely acknowledged that increased prevalence of skin cancer in the elder population is primarily caused by accumulated DNA damage that is left unrepaired by overwhelmed and less efficient genomic maintenance machinery, which ultimately lead to harmful mutations. Recent evidences imply the additional possibility that mechanical properties of the aging skin, including altered stiffness, elastic modulus, and shear modulus, significantly contribute to the accelerated pace of tumor progression.
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