Abstract
Cutaneous squamous cell carcinoma (cSCC) is the second most common skin cancer and actinic keratosis (AK) is one of the precancerous lesion of cSCC. It is necessary to control the development because there are not enough treatments established for advanced cSCC. Most cases of AK are restricted to the epidermis for a long time through the suppression of epithelial-to-mesenchymal transition (EMT). While ovo like transcriptional repressor 1 (OVOL1) and ovo like zinc finger 2 (OVOL2) are important modulators of EMT in some tumors, little is known in skin tumors.
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