Abstract
A Congenital anomaly of the portal vein characterized by the clinical sign of ascites was diagnosed in a dog with a marked increase in blood ammonia (490μg/dl) and BSP retention (51.4%). Total serum protein value was within normal limits. Neither sodium accumulation nor hypoalbuminemia was found; nor were ammonium biurate crystals found in the urine. Angiographically, anastomosis of the portal vein with the azygous vein and retrograde flow from the portal vein to the gastric vein were detected. At necropsy, the latter was confirmed to be of gastroesophageal collateral origin carrying blood from the portal vein not into the liver but into the azygous vein. In this study, it was suspected that congenital absence of a hepatoportal system led to the development of acquired gastroesophageal collaterals and ascites originating from the marked portal hypertension.
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