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Abstract
Effective inflammatory responses arise from coordinated reaction of diverse immune cell populations to exogenous stimuli. The considerable heterogeneity of these populations has impeded high-resolution dissection of their molecular behavior during activation. To systematically characterize cutaneous inflammatory response, we analyzed 52,086 immune cells isolated from mouse skin by single cell RNA sequencing (scRNA-seq), after induction of Toll-like receptor (TLR)-dependent inflammation using imiquimod (IMQ) or delayed-type hypersensitivity using oxazolone (OXA).
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