Abstract
Alopecia Areata (AA) is a common autoimmune disease characterized by T cell infiltration of the hair follicle, resulting in nonscarring hair loss. Although CD4 T cells comprise the majority of the infiltrate, little is known about their contribution to disease pathogenesis. Previously we have identified an increased number of IFN-γ producing CD4+ T cells in the skin-draining lymph nodes (SDLN) as compared to unaffected (UA) controls. The objective for this study was to determine the role that CD4 T cells play in the pathogenesis of AA.
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