Abstract
Atopic dermatitis (AD) is a chronic inflammatory skin disease characterized by a dysregulated immune response associated with barrier dysfunction and dysbiosis of the skin microbiota characterized by Staphylococcus aureus colonization in lesional skin. The aim of our study was to compare non-lesional and lesional AD skin transcriptional profile depending on staphylococcal colonization. Skin biopsies were analyzed by RT-qPCR in moderate and severe adults AD patients. Staphylococcus epidermidis was detected in all lesional skin, in greater abundance than in non- lesional skin, while S.
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