Abstract
Vitiligo is a relatively common, acquired depigmenting disease caused by the loss of epidermal melanocytes. Oxidative stress is known to participate in the initiation of vitiligo by triggering autoimmunity. In this study, we tried to identify factors serving as a bridge between oxidative stress and autoimmunity in vitiligo. According to RNA sequencing analysis on H2O2-treated melanocytes, interferon-stimulated gene 15 (ISG15) among interferon signaling-related genes were particularly upregulated.
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Published version (
Free)
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
