Abstract

Genital sexual arousal, which consists of genital vasocongestion and lubrication, is critical to healthy sexual function in women and closely linked with hormone function. Oral hormonal contraceptive pills (OCPs), which are used by over a quarter of reproductive-age women in the United States, tend to reduce the number of bioavailable androgens and have been associated with decrements in self-reported arousal and vaginal lubrication. However, empirical examinations of the possible physiological effects of OCPs on women’s sexual arousal response are sparse.

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