Abstract

One of the essential points of cardiac rehabilitation is to understanding the exercise physiology of exercise-induced metabolic changes through the study of the acute responses and chronic adaptation to exercise. Exercise capacity is expressed as maximal oxygen consumption (VO2max) and its unit is expressed as the amount of oxygen consumed per kilogram of body weight per minute (mL/kg/min). VO2 can be calculated as stroke volume x heart rate x arteriovenous oxygen difference, and in cardiac rehabilitation, the arteriovenous oxygen difference should be maximized. Oxygen consumption during exercise is expressed as a multiple of oxygen consumption during rest. Metabolic equivalent (MET) is the basic metabolic energy consumed during rest, which is 3.5 mL/kg/min. Myocardial oxygen uptake (MVO2) is determined by myocardial wall tension, contractility, and heart rate. It does not increase further when the angina threshold is reached. Aerobic training can lower MVO2 at rest and at submaximal exercise intensity. As a result, when performing the same intensity of activity, MVO2 is lowered and the margin of safety is widened, and preventing from occurring at the same intensity or higher intensity compared to the past. It is very useful to know each individual

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