Abstract
Cardiac disruption and development of a diabetic cardiomyopathy (DCM) are a direct consequence of diabetes. In work activity Na/K-ATPase in a myocardium of rats with different extent of diabetic impairment was studied. Suppression of activity of Na/K-ATPase in the cardiomyocytes the left ventricle of rats was revealed at strongly developed diabetes, but not at a prediabetes stage. It was concluded, that development of DCM breaks transport processes in a membrane of rats cardiomyocytes, and the expressiveness of observed changes depends on diabetes degree.
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