Abstract
Parvovirus infection B19 is a pressing problem in pediatrics due to its widespread distribution and polymorphism of clinical manifestations. At present, the causes of damage to the nervous system during acute parvovirus B19 infection are not completely clear, although the virus is able to persist in neuroglial cells only in immunocompromised individuals and is not neurotropic. The article presents two clinical observations of neuroinfections caused by Parvovirus B19 infection. In one clinical case, severe damage to the brain substance with the development of convulsive syndrome was described. Timely administration of complex etiopathogenetic therapy contributed to a favorable outcome, despite the severity of the disease. A special feature of the second clinical case was the two-wave course of parvovirus infection B19 followed by the development of acute serous viral meningitis of combined etiology (parvovirus B19 + tick-borne encephalitis virus). In both cases, along with etiotropic treatment, a complex drug cytoflavin was used as pathogenetic therapy, which helps optimize the course of the disease and favorable outcomes. Key words: parvovirus B19, neuroinfections, encephalitis, meningitis, children, cytoflavin
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