장시간의 고강도 유산소 운동이 항산화 효소 및 근 손상지표에 미치는 영향

Abstract

Purpose: The purpose of this study is to investigate the effects of long-term aerobic exercise on muscle damage indicators and antioxidant enzymes by observation of amateur athlete participants. We observed the changes that occurred at rest and immediately after a marathon and triathlon.<BR> Method: The subjects of the study were 8 amateur male marathon runners and 9 amateur triathletes. All participants were in their 30’s and 40’s of age and resided in the city of S. After the marathon runners ran a full race of 42.195 kilometers, two blood samples were taken immediately after the race when their conditions were stable. After the triathletes went through an Olympic course which included 1.5 kilometers of swimming, 40 kilometers of cycling, and 10 kilometers of running, two blood samples were also taken immediately after completion when their conditions were stable. Using the SPSS/PC+Ver. 21.0K program, descriptive statistics (Mean ± Standard Error) of all dependent variables were calculated. A two-way repeated ANOVA was performed to verify the difference between the average of the dependent variables once the homogeneity between the two groups (marathon and triathlon) and between thew to tests (stable and immediately after the race) were confirmed. A paired t-test was performed for the differences of the two tests within the groups, and an independent t-test was performed for the differences between the two groups. The statistical significance level was set to α=.05.<BR> Results: In the marathon group, SOD significantly increased immediately after the race (p<.001). In the triathlon group, SOD significantly increased in the same manner after the race (p<.05). There was no significant difference between the two groups. In both groups, CK and LDHi significantly increased immediately after the races (p<.001), with no significant differences between the groups.<BR> Conclusion: During long-term high-intensity aerobic exercise, marathons and triathlons increase oxidative stress, which can lead to muscle damage. Additionally, the increase of SOD, an antioxidant defense system enzyme, is a response to oxidative stress and appears to prevent the generation of free radicals from exercise.