Abstract
In this study examined the effects of a multivalent protease inhibitor aprotininum on the state indicators of blood coagulation and antiproteinase potential during the development of experimental reperfusion syndrome, as well as the severity of morphological changes in liver tissue. It was found that with a 12-hour reperfusion syndrome in the background the development of an imbalance in the proteinase-inhibitor system occurs coagulopathy consumption and there are initial liver damage with development necrosis and plethora of blood vessels. Against the background of the use of aprotininum with experimental reperfusion syndrome is less marked severe liver damage decreases imbalance in condition of non-specific proteinases and their inhibitors without deeper disorders in the blood coagulation system.
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