Post-traumatic Subdural Hygroma Research Articles

Posttraumatic arachnoid cyst rupturę and delayed acute subdural hygroma

Introduction. Subdural hygromas (SDGs) are the accumulation of fluid in the subdural space. Arachnoid cysts (ACs) on the other hand are common cerebrospinal fluid (CSF) containing lesions of the central nervous system, located within the subarachnoid space. They are generally found incidentally on neuroimaging studies and remain asymptomatic throughout the life. Rupture into the subdural space resulting in subdural hygroma (SDG) is relatively rare. Aim. We aimed to show the importance of the radiological follow up in head trauma patients having large ACs. Description of the case. We report a case of a 69-year-old male patient with a known large Galassi type III AC, presented to our hospital with traumatic brain injuries and re-presented with acute posttraumatic SDG in association with AC rupture. Conclusion. This case emphasizes the importance of radiological follow up in head trauma patients having large ACs to reveal and appropriately manage traumatic subdural collections.

Post-traumatic subdural hygroma: About two cases

Post-traumatic subdural hygroma: About two cases

Post-Traumatic Subdural Hygroma: A One Year Follow up Case Report and Literature Review

<p>Post-traumatic subdural hygroma it is an intracranial lesion, commonly misdiagnosed due to its unclear symptomatology and slow progression. Defined as the presence of cerebral spinal fluid in between the arachnoid and the duramater layers; causing a variety of symptomatology that usually depend on the amount of fluid within this space (subdural). Post traumatic subdural hygroma it’s observed in 5-20% of all blunt head injuries, although its diagnosis it’s usually rare among emergency departments. Prognosis is usually good due to the absence of severe complications and its spontaneous resolution in mostly of the cases. We present a 26-year-old male patient, with history of high impact blunt trauma, presenting ocular refractive changes and headache. After CT scan evaluation and BOLD-MRI confirmation, a bilateral fronto-parietal subdural hygroma was confirmed.</p>

The study of the improved complex neurosurgical treatment in patients with posttraumatic chronic subdural hematomas and hygromas

The study of the improved complex neurosurgical treatment in patients with posttraumatic chronic subdural hematomas and hygromas

The study of the improved complex neurosurgical treatment in patients with posttraumatic chronic subdural hematomas and hygromas

The study of the improved complex neurosurgical treatment in patients with posttraumatic chronic subdural hematomas and hygromas

Advances in diagnosis and treatment of posttraumatic subdural hygroma

Advances in diagnosis and treatment of posttraumatic subdural hygroma

Pediatric post-traumatic subdural hygroma associated with a giant frontotemporal arachnoid cyst

We report a rare case of a post-traumatic subdural hygroma occurring with a giant frontotemporal arachnoid cyst in a 6-year old child and discuss its pathogenesis and management implications. Awareness regarding the association of a subdural hygroma associated with an arachnoid cyst is of immense clinical importance as the former may precipitate intracranial hypertension in a previously asymptomatic arachnoid cyst and yet may remain undetectable on a CT scan.

Post-traumatic subdural hygroma: Case report

Abstract Subdural Hygroma (SDG) is described in the literature as a common complication of blunt head trauma occurring in a reported 5–20% of all closed head injuries ( 1–3). The Emergency Physician will likely encounter this type of lesion and it is useful to have an understanding of its significance, its relationship to subdural hematoma (SDH), and management strategies. Although surgical intervention is often advocated in the treatment of intracranial fluid collections causing mass effect, there are several reported cases of spontaneous resolution of uncal and central herniation via conservative management alone ( 4,5). The authors report a case of a 90-year-old woman who presented to the Emergency Department 5 days after falling and sustaining a closed head injury with computed tomography (CT) scan evidence of mid-line shift.

Elevated cerebral perfusion pressure and low colloid osmotic pressure as a risk factor for subdural space-occupying hygromas?

BACKGROUND Space-occupying subdural hygromas are a late complication of severe traumatic brain injury (TBI) and may delay the patient’s recovery. To evaluate the risk factors involved, we performed a semiretrospective, -prospective analysis of three groups of patients, which differed with regard to the techniques used in the management of their cerebral perfusion pressure (CPP) and colloid osmotic pressure (COP) to determine the occurrence of space-occupying subdural hygromas. PATIENTS AND METHODS Between 1989 and 1997 we examined 696 patients after a severe TBI: Group 1. 1989–1994 mean CPP: 67 (elevated for therapeutic reasons by catecholamines, if necessary), mean COP: 19. Group 2. January 1995–October 1996, mean CPP: 77, mean COP: 20. Group 3. November 1996–December 1997, mean CPP: 79, mean COP: 23 (elevated for therapeutic reasons by infusions of colloids). The groups were comparable for other criteria. RESULTS Compared to Group 1, Group 2, with a high CPP but lower COP, showed a significantly higher (p < 0.01; χ 2-test with correction of Yates) percentage of posttraumatic subdural hygromas with space-occupying aspects, clinical signs of bradycardia, hypertension and impaired consciousness requiring surgery (Group 1: 1.75%; Group 2: 10.46%; Group 3: 0%). In Group 3 we saw no patient with a space-occupying hygroma. CONCLUSION We conclude that iatrogenic elevated CPP, which has been reported to be helpful in preventing secondary ischemic damage after a severe TBI, may be harmful to a patient if the COP is not maintained within physiological ranges.

Dynamics and pathogenesis of traumatic subdural hygroma

Of 2005 patients, who had suffered blunt skull trauma, 108 developed post-traumatic subdural hygromas (TSH). CT was used to observe the course of subdural hygromas not treated surgically, with particular reference to the time of their appearance and regression. The relative incidence of TSH, and of the severity of head trauma quantified on a 5-grade scale was determined in different age groups. From the traumatic changes demonstrated on CT, some conclusions could be drawn regarding the pathogenesis. A hypothetical model has been developed which explains TSH as a result of shearing forces.

Protein analysis of subdural hygroma fluid.

The pathogenesis of posttraumatic subdural hygroma still remains largely unknown. One of the suggested pathological mechanisms is the traumatic development of an arachnoid tear and the subsequent efflux of CSF into the subdural space. We performed a multifactorial analysis of the hygroma fluid obtained at operation in comparison to the simultaneously taken plasma and lumbar CSF. The results of the protein analysis support the CSF origin of the subdural hygroma fluid.

A Case of Bilateral Chronic Subdural Hematoma Secondary to Posttraumatic Subdural Hygroma

A Case of Bilateral Chronic Subdural Hematoma Secondary to Posttraumatic Subdural Hygroma

The clinical course of surgically treated posttraumatic subdural hygromas.

The clinical course of 11 patients with a subdural hygroma, who underwent neurosurgery, was studied. In the first week after the operation no improvement of the mental status could be detected. We considered therefore that operation hardly contributed to improvement. Thus the need to operate is limited to special cases such as acute or space-occupying subdural hygroma.

Traumatic Subdural Hygroma in Adults

The authors review 25 cases of post-traumatic subdural hygroma in adults and report the findings of computer tomography in 4 cases. There was marked diversity in the severity of injury and in chronicity. The site of the lesion was consistently delineated by angiography, but preoperative differentiation from subdural hematoma was possible only with computed tomography. The mortality was 28%. Good recovery occurred in 48%. The prognosis was best among younger patients with subacute or chronic lesions.

Acute, Post-Traumatic Subdural Hygroma. Natural Course with Angiographic Documentation.

Acute, post-traumatic subdural hygromas, in contrast to benign chronic subdural hygromas, may be life-threatening. Two cases are presented in which a rapidly enlarging subdural hygroma either prevented improvement over a 1-2 week period or resulted in rapid deterioration of the patient. Angiographic documentation and the natural course of this condition are revealed.