Journal Of Clinical Investigation Research Articles

FURTHER OBSERVATION ON POST‐TRANSFUSION PURPURA (PTP)

British Journal of HaematologyVolume 61, Issue 2 p. 374-375 Free Access FURTHER OBSERVATION ON POST-TRANSFUSION PURPURA (PTP) A. E. G. Kr. Von Dem Borne, A. E. G. Kr. Von Dem Borne Department of Immunohaematology, Central Laboratory of the Netherlands Red Cross Blood Transfusion Service and Department of Haematology, Academic Medical Centre, University of Amsterdam, P.O. Box 9406, Amsterdan, The NetherlandsSearch for more papers by this authorC. M. Van Der Plas-van Dalen, C. M. Van Der Plas-van Dalen Department of Immunohaematology, Central Laboratory of the Netherlands Red Cross Blood Transfusion Service and Department of Haematology, Academic Medical Centre, University of Amsterdam, P.O. Box 9406, Amsterdan, The NetherlandsSearch for more papers by this author A. E. G. Kr. Von Dem Borne, A. E. G. Kr. Von Dem Borne Department of Immunohaematology, Central Laboratory of the Netherlands Red Cross Blood Transfusion Service and Department of Haematology, Academic Medical Centre, University of Amsterdam, P.O. Box 9406, Amsterdan, The NetherlandsSearch for more papers by this authorC. M. Van Der Plas-van Dalen, C. M. Van Der Plas-van Dalen Department of Immunohaematology, Central Laboratory of the Netherlands Red Cross Blood Transfusion Service and Department of Haematology, Academic Medical Centre, University of Amsterdam, P.O. Box 9406, Amsterdan, The NetherlandsSearch for more papers by this author First published: October 1985 https://doi.org/10.1111/j.1365-2141.1985.tb02838.xCitations: 27AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL No abstract is available for this article. References Pegels, J.G., Bruynes, E.C.E., Engelfriet, C.P. & von dem Borne, A.E.G. Kr. (1981) Post-transfusion purpura: a serological and immunochemical study. British Journal of Haematology, 49, 521– 530. Boissard, B. & Wautier, J.L. (1984) Leka, a new platelet antigen absent in Glanzmann's throm-basthenia. Vox Sanguinis, 46, 47– 54. Shulman, N.R., Aster, R.H., Leitner, A. & Hiller, M.C. (1961) Immunoreaction involving platelets. V. Posttransfusion purpura due to a complement-fixing antibody against a genetically controlled platelet antigen. A proposed mechanism for thrombocytopenia and its relevancy in ‘autoimmunity’. Journal of Clinical Investigation, 40, 1597– 1620. Keimowitz, R.M., Collins, J., Davis, K. & Aster, R.H. (1985) Post-transfusion purpura associated with alloimmunization against the platelet-specific antigen, Baka. (In press). Citing Literature Volume61, Issue2October 1985Pages 374-375 ReferencesRelatedInformation

RED CELL ENZYME ACTIVITIES IN THALASSAEMIA

British Journal of HaematologyVolume 57, Issue 4 p. 711-712 Free Access RED CELL ENZYME ACTIVITIES IN THALASSAEMIA Barbara B. Anderson, Barbara B. Anderson Department of Haematology, St Bartholomew's Hospital, London EC1A 7BESearch for more papers by this authorGillian M. Perry, Gillian M. Perry Department of Haematology, St Bartholomew's Hospital, London EC1A 7BESearch for more papers by this authorJanet E. Clements, Janet E. Clements Department of Haematology, St Bartholomew's Hospital, London EC1A 7BESearch for more papers by this author Barbara B. Anderson, Barbara B. Anderson Department of Haematology, St Bartholomew's Hospital, London EC1A 7BESearch for more papers by this authorGillian M. Perry, Gillian M. Perry Department of Haematology, St Bartholomew's Hospital, London EC1A 7BESearch for more papers by this authorJanet E. Clements, Janet E. Clements Department of Haematology, St Bartholomew's Hospital, London EC1A 7BESearch for more papers by this author First published: August 1984 https://doi.org/10.1111/j.1365-2141.1984.tb02950.xCitations: 3AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinkedInRedditWechat No abstract is available for this article. References Anderson, B.B., Fulford-Jones, C.E., Child, J.A., Beard, M.E.J. & Bateman, C.J.T. (1971) Conversion of vitamin B6 compounds to active forms in the red blood cell. Journal of Clinical Investigation, 50, 1901– 1909. Anderson, B.B. & Mollin, D.L. (1972) Red-cell metabolism of pyridoxine in sideroblastic anaemias and related anaemias. British Journal of Haematology, 23, (Suppl.), 159– 166. Anderson, B.B., Mollin, D.L., Child, J.A., Modell, C.B. & Perry, G.M. (1975) Red-cell metabolism of pyridoxine in sideroblastic anaemia and thalassaemia. Excerpta Medica (International Congress Series 366), 241– 250. Anderson, B.B., Perry, G.M., Modell, C.B., Child, J.A. & Mollin, D.L. (1979) Abnormal red-cell metabolism of pyridoxine associated with β-thalassaemia. British Journal of Haematology, 41, 497– 507. Chern, C.J. & Beutler, E. (1976) Biochemical and electrophoretic studies of erythrocyte pyridoxine kinase in white and black Americans. American Journal of Human Genetics, 28, 9– 17. Chillar, R.K., Johnson, C.S. & Beutler, E. (1976) Erythrocyte pyridoxine kinase levels in patients with sideroblastic anaemia. New England Journal of Medicine, 295, 881– 883. Clements, J.E. & Anderson, B.B. (1980) Pyridoxine (pyridoxamine) phosphate oxidase activity in the red cell. Biochimica et Biophysica Acta, 613, 401– 409. Clements, J.E., Anderson, B.B. & Perry, G.M. (1981) Low red cell activity of pyridoxine (pyridoxamine) phosphate oxidase and glutathione reductase associated with thalassaemia. Biomedicine, 34, 119– 123. Hamfelt, A. (1967) Pyridoxal kinase activity in blood cells. Clinica Chimica Acta, 16, 7– 18. Lumeng, L. & Li, T.K. (1974) Vitamin B6 metabolism in chronic alcohol abuse. Pyridoxal phosphate levels in plasma and the effects of acetaldehyde on pyridoxal phosphate synthesis and degradation in human erythrocytes. Journal of Clinical Investigation, 53, 693– 704. Solomon, L.R. & Hillman, R.S. (1978) Vitamin B6 metabolism in human red cells. 1. Variation in normal subjects. Enzyme, 23, 262– 273. Solomon, L.R. & Hillman, R.S. (1979) Vitamin B6 metabolism in idiopathic sideroblastic anaemia and related disorders. British Journal of Haematology, 42, 239– 253. Vives Corrons, J.L., Pujades, M.A., Aguilar, I, Bascompte, J.L, Jou, J.M., Rozman, C. & Ester, A. (1984) Pyrimidine 5′nucleotidase and several other red cell enzyme activities in β-thalassaemia trait. British Journal of Haematology, 56, 483– 494. Yamada, K. & Tsuji, M. (1966) Transport of vitamin B6 in human erythrocytes (III). The effect of phosphate on transport of vitamin B6 in human erythrocyte. Vitamins (Japan), 33, 35– 38. Citing Literature Volume57, Issue4August 1984Pages 711-712 ReferencesRelatedInformation

Glutamine production rate and its contribution to urinary ammonia in normal man.

1. Glutamine [15N]amide was infused at a steady rate of 33.34 mumol/h into seven male adult volunteers who were in the fed state and normal acid-base status. 2. Plasma glutamine amide N enrichment and urinary ammonia N enrichment rose to a constant rate within 3 h. 3. The glutamine production rate was 51.8 +/- 7.9 mmol/h. 4. The total ammonia excretion rate was 0.87 mmol/h. Of this excreted ammonia 62.6 +/- 9% was derived from the amide N atom of glutamine. 5. The excreted glutamine amide N (0.53 mmol/h) was only 1% of the glutamine production. If half the ammonia formed by the kidney is excreted in urine and half liberated into the renal vein in subjects with normal acid-base status [E. E. Owen & R. R. Robinson (1963) Journal of Clinical Investigation, 42, 263-276], then the kidney accounts for only 2% of glutamine disposal. 6. Whole body protein turnover, measured from the urinary [15N]ammonia enrichment, was 30.3 +/- 7.7 g of N/day (2.8 g of protein day-1 kg-1).

Experimental Hematology Today 1981

This volume represents a new in scientific publication: an investigative journal in the guise of a book; for there is little, apart from the hard cover, to distinguish this volume from an issue of Experimental Hematology or other investigative journals such as the Journal of Clinical Investigation . The volume contains 26 papers (by 104 authors), all purely investigative in both format and content. The genre has multiplied in recent years, apparently through the intensification of the publish or perish dictum. The content is the editors' selection of some of the papers presented at the ninth annual meeting of the International Society for Experimental Hematology held in Dallas in 1980. The bases for selection are not clear. Similar selections from previous meetings of the society, bearing the same title, have been published during the past few years. The papers are divided into six parts: a presidential symposium without a theme,

Role of the renin-angiotensin system in prostaglandin E2-induced hypertension.

HomeHypertensionVol. 2, No. 4Role of the renin-angiotensin system in prostaglandin E2-induced hypertension. Free AccessAbstractPDF/EPUBAboutView PDFSections ToolsAdd to favoritesDownload citationsTrack citationsPermissions ShareShare onFacebookTwitterLinked InMendeleyReddit Jump toFree AccessAbstractPDF/EPUBRole of the renin-angiotensin system in prostaglandin E2-induced hypertension. G M Hockel and A W CowleyJr G M HockelG M Hockel Search for more papers by this author and A W CowleyJrA W CowleyJr Search for more papers by this author Originally published1 Jul 1980https://doi.org/10.1161/01.HYP.2.4.529Hypertension. 1980;2:529–537 Previous Back to top Next FiguresReferencesRelatedDetailsCited By Liu B, Wu X, Zeng R, Yin Y, Guo T, Xu Y, Zhang Y, Leng J, Ge J, Yu G, Guo J and Zhou Y (2019) Prostaglandin E 2 sequentially activates E‐prostanoid receptor‐3 and thromboxane prostanoid receptor to evoke contraction and increase in resistance of the mouse renal vasculature , The FASEB Journal, 10.1096/fj.201901611R, 34:2, (2568-2578), Online publication date: 1-Feb-2020. Stock J, Shinjo K, Burkhardt J, Roach M, Taniguchi K, Ishikawa T, Kim H, Flannery P, Coffman T, McNeish J and Audoly L (2001) The prostaglandin E2 EP1 receptor mediates pain perception and regulates blood pressure, Journal of Clinical Investigation, 10.1172/JCI6749, 107:3, (325-331), Online publication date: 1-Feb-2001. Wang J, Cheng H and Harris R (1999) Cyclooxygenase-2 Inhibition Decreases Renin Content and Lowers Blood Pressure in a Model of Renovascular Hypertension, Hypertension, 34:1, (96-101), Online publication date: 1-Jul-1999. Audoly L, Tilley S, Goulet J, Key M, Nguyen M, Stock J, McNeish J, Koller B and Coffman T (1999) Identification of specific EP receptors responsible for the hemodynamic effects of PGE 2 , American Journal of Physiology-Heart and Circulatory Physiology, 10.1152/ajpheart.1999.277.3.H924, 277:3, (H924-H930), Online publication date: 1-Sep-1999. Tilley S, Audoly L, Hicks E, Kim H, Flannery P, Coffman T and Koller B (1999) Reproductive failure and reduced blood pressure in mice lacking the EP2 prostaglandin E2 receptor, Journal of Clinical Investigation, 10.1172/JCI6579, 103:11, (1539-1545), Online publication date: 1-Jun-1999. Cowley A, Skelton M and Merrill D (1986) Are hypertensive effects of aldosterone, angiotensin, vasopressin, and norepinephrine chronically additive?, Hypertension, 8:4, (332-343), Online publication date: 1-Apr-1986. Boudier H (1984) The interaction of antihypertensive drugs with mechanisms of blood pressure regulation Pharmacology of Antihypertensive Drugs, 10.1016/B978-0-444-90313-6.50009-5, (46-65), . HAYASHI M, FLUITA A and SATOH S (1983) DIFFERENT MODE OF ACTION BETWEEN NOREPINEPHRINE AND PHENYLEPHRINE ON PROSTAGLANDIN SYNTHESIS BY DOG RENAL INNER MEDULLARY SLICES, Japanese Journal of Pharmacology, 10.1016/S0021-5198(19)52492-1, 33:3, (563-571), . July 1980Vol 2, Issue 4 Article InformationMetrics Copyright © 1980 by American Heart Associationhttps://doi.org/10.1161/01.HYP.2.4.529PMID: 6995293 Originally publishedJuly 1, 1980 PDF download Advertisement

Osmometry. 2. Osmoregulation.

The maintenance of osmolar constancy of the body fluids is dependent upon the recognition of osmolar disequilibrium and its correction by modifying the ingestion and excretion of fluid and solute. Osmolar changes are sensed by the hypothalamus which regulates the secretion of antidiuretic hormone to modify the renal excretion of water. The integrity of the system depends upon the renal ability to vary the solute concentration of urine.

Principal substrates of fetal metabolism.

Principal substrates of fetal metabolism.

ADENOSINE DEAMINASE ACTIVITY IN LYMPHOCYTES

British Journal of HaematologyVolume 37, Issue 1 p. 157-158 Free Access ADENOSINE DEAMINASE ACTIVITY IN LYMPHOCYTES J. L. Sullivan, J. L. Sullivan Department of Pediatrics, University of Washington School of Medicine, Seattle, Washington 98195, U.S.A.Search for more papers by this authorW. R. A. Osborne, W. R. A. Osborne Department of Pediatrics, University of Washington School of Medicine, Seattle, Washington 98195, U.S.A.Search for more papers by this authorR. J. Wedgwood, R. J. Wedgwood Department of Pediatrics, University of Washington School of Medicine, Seattle, Washington 98195, U.S.A.Search for more papers by this author J. L. Sullivan, J. L. Sullivan Department of Pediatrics, University of Washington School of Medicine, Seattle, Washington 98195, U.S.A.Search for more papers by this authorW. R. A. Osborne, W. R. A. Osborne Department of Pediatrics, University of Washington School of Medicine, Seattle, Washington 98195, U.S.A.Search for more papers by this authorR. J. Wedgwood, R. J. Wedgwood Department of Pediatrics, University of Washington School of Medicine, Seattle, Washington 98195, U.S.A.Search for more papers by this author First published: September 1977 https://doi.org/10.1111/j.1365-2141.1977.tb08825.xCitations: 49AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinkedInRedditWechat No abstract is available for this article. References Fu, S.M., Winchester, R.J. & Kunkel, H.G. (1975) The occurrence of the HL-B alloantigens on the cells of unclassified acute lymphoblastic leukemias. Journal of Experimental Medicine, 142, 1334. Kaplan, J., Ravindranath, Y. & Peterson, W.D. (1977) T and B lymphocyte antigen-positive null cell leukemias. Blood, 49, 371. Nilsson, K. (1976) Establishment of permanent human lymphoblastoid cell lines in vitro. In Vitro Methods in Cell Mediated and Tumor Immunity, p 713. Academic Press, London . Ramot, B., Brok-Simoni, F., Barnea, N., Banik, I. & Holtzman, F. (1977) Adenosine deaminase (ADA) activity in lymphocytes of normal individuals and patients with chronic lymphatic leukaemia. British Journal of Haematology, 36, 67. Royston, I., Pitts, R.B., Smith, R.W. & Graze, P.R. (1975) In vitro immunization against cultured human B and T lymphoblastoid cell lines. Transplantation Proceedings, 7 (Suppl. 1), 531. Tung, R., Silber, R., Quagliata, F., Conklyn, M., Gottesman, J. & Hirschhorn, R. (1976) Adenosine deaminase activity in chronic lymphocytic leukemia. Journal of Clinical Investigation, 57, 756. Citing Literature Volume37, Issue1September 1977Pages 157-158 ReferencesRelatedInformation

Arnold S. Relman, editor-elect of the Journal.

No extract is available for articles shorter than 400 words.

The value to medical practice of basic and applied medical research done twenty years ago.

Research can be justified in many ways besides the improvement of clinical practice--for example, by providing the intellectual excitement necessary for teachers and students. The value of present-day research cannot be assessed; but that of twenty years ago can be, at least in respect of current medical practice. An analysis has been made of all the papers published in 1956 in the American Journal of Physiology, the Journal of Physiology (London), the Journal of Clinical Investigation, and Clinical Science. This shows an overwhelming impact on current medical practice coming from clinical departments, and a comparatively very small influence of work coming from preclinical departments. If this conclusion is correct and generally applicable, it has some relevance to the funding of medical research.

Ideas and anomalies in the evolution of modern oxygen therapy.

Ideas and anomalies in the evolution of modern oxygen therapy.

Interaction of thermal and baroreceptor reflexes in man.

Interaction of thermal and baroreceptor reflexes in man.

Carrier-mediated blood-brain barrier transport of short-chain monocarboxylic organic acids

Carrier-mediated blood-brain barrier transport of short-chain monocarboxylic organic acids

Transductal fluxes of Na, K, and water in the human eccrine sweat gland.

ARTICLESTransductal fluxes of Na, K, and water in the human eccrine sweat glandJ MangosJ MangosPublished Online:01 May 1973https://doi.org/10.1152/ajplegacy.1973.224.5.1235MoreSectionsPDF (1 MB)Download PDF ToolsExport citationAdd to favoritesGet permissionsTrack citations ShareShare onFacebookTwitterLinkedInWeChat Previous Back to Top Download PDF FiguresReferencesRelatedInformation Cited ByHuman temperature regulation under heat stress in health, disease, and injuryMatthew N. Cramer, Daniel Gagnon, Orlando Laitano, and Craig G. Crandall19 August 2022 | Physiological Reviews, Vol. 102, No. 4Method for Human Eccrine Sweat Gland Isolation from the Scalp by Means of the Micropunch Technique and Vital Dyes21 April 2020An efficient method for eccrine gland isolation from human scalp3 April 2018 | Experimental Dermatology, Vol. 27, No. 6Sweating as a heat loss thermoeffectorComprehensive and Quantitative Profiling of the Human Sweat Submetabolome Using High-Performance Chemical Isotope Labeling LC–MS8 July 2016 | Analytical Chemistry, Vol. 88, No. 14Abolished InsP3R2 function inhibits sweat secretion in both humans and mice20 October 2014 | Journal of Clinical Investigation, Vol. 124, No. 11From frog integument to human skin: dermatological perspectives from frog skin biology3 December 2013 | Biological Reviews, Vol. 89, No. 3Hot bath for the treatment of chronic renal failure24 September 2013 | Renal Failure, Vol. 36, No. 1Regional variations in transepidermal water loss, eccrine sweat gland density, sweat secretion rates and electrolyte composition in resting and exercising humans1 February 2013 | Extreme Physiology & Medicine, Vol. 2, No. 1Changes in Na+, K+ concentrations in perspiration and perspiration volume with alternating current iontophoresis in palmoplantar hyperhidrosis patients2 August 2008 | Archives of Dermatological Research, Vol. 300, No. 10Na+ secretion rate increases proportionally more than the Na+ reabsorption rate with increases in sweat rateMichael J. Buono, Ryan Claros, Teshina DeBoer, and Janine Wong1 October 2008 | Journal of Applied Physiology, Vol. 105, No. 4Na+/H+ exchangers in the human eccrine sweat ductD. Granger, M. Marsolais, J. Burry, and R. Laprade1 November 2003 | American Journal of Physiology-Cell Physiology, Vol. 285, No. 5Effects of non-pharmacological sympathetic sudomotor denervation on sweating in humans with essential palmar hyperhidrosisClinical Biochemistry, Vol. 30, No. 2An analysis on the rates and regulation of insensible water loss through the eccrine sweat glandsJournal of Theoretical Biology, Vol. 141, No. 3Biology of sweat glands and their disorders. I. Normal sweat gland functionJournal of the American Academy of Dermatology, Vol. 20, No. 4The pathogenesis of cystic fibrosis: A proposal for calmodulin as the basic biochemical defectMedical Hypotheses, Vol. 20, No. 3Effects of some ion transport inhibitors on secretion and reabsorption in intact and perfused single human sweat glandsPflügers Archiv - European Journal of Physiology, Vol. 391, No. 4Studies on the basic defect in cystic fibrosis: The kinin hypothesis1 December 1978 | Journal of Inherited Metabolic Disease, Vol. 1, No. 4EXOCRINE-GLAND FUNCTION AND THE BASIC BIOCHEMICAL DEFECT IN CYSTIC FIBROSISThe Lancet, Vol. 312, No. 8086Cystic FibrosisThe Sweat GlandsEpithelial transport of potassiumKidney International, Vol. 11, No. 6The physiology, pharmacology, and biochemistry of the eccrine sweat gland22 June 2005 More from this issue > Volume 224Issue 5May 1973Pages 1235-1240 Copyright & PermissionsCopyright © 1973 by American Physiological Societyhttps://doi.org/10.1152/ajplegacy.1973.224.5.1235PubMed4700643History Published online 1 May 1973 Published in print 1 May 1973 Metrics

Anaesthesia in sickle cell states.

AnaesthesiaVolume 28, Issue 3 p. 339-341 Free Access Anaesthesia in sickle cell states T. Hilary Howells, T. Hilary Howells The Royal Free Hospital, North Western Branch, London NW3 2XJSearch for more papers by this authorR. G. Huntsman, R. G. Huntsman The Royal Free Hospital, North Western Branch, London NW3 2XJSearch for more papers by this author T. Hilary Howells, T. Hilary Howells The Royal Free Hospital, North Western Branch, London NW3 2XJSearch for more papers by this authorR. G. Huntsman, R. G. Huntsman The Royal Free Hospital, North Western Branch, London NW3 2XJSearch for more papers by this author First published: May 1973 https://doi.org/10.1111/j.1365-2044.1973.tb00464.xCitations: 4 Editorial note. It is regretted that this letter was received too late for us to obtain a reply from Dr Searle for this issue. Dr Searle was in transit at the time his proofs were available for correction and we therefore regret two errors of fact which remained in the published version of his paper. Page 49, line 15 should read ‘valine for glutamic acid’ not ‘valine for glutamine’. Page 51, last paragraph ‘glutamic acid by lysine’ not ‘glutamine by lysine’. AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL References 1 Green, R.L., Huntsman, R.G. & Serjeant, G.R (1972) Sickle-cell and altitude. British Medical Journal, 1, 803. 2 Konotey-Ahulu, F.I.D. (1969) Anaesthetic deaths and the sickle-cell trait. Lancet, i, 267. 3 Neel, J.V., Wells, I.C. & Itano, H.A. (1951) Familial differences in the proportion of abnormal hemoglobin present in the sickle-cell trait. Journal of Clinical Investigation, 30, 1120. 4 McLellan, D.S., Huntsman, R.G. & Laidler, P. (1973) In preparation. 5 Howells, T.H., Huntsman, R.G., Boys, J.E. & Mahmood, A. (1972) Anaesthesia and sickle-cell haemoglobin. British Journal of Anaesthesia, 44, 975. 6 Odunton, S.A. & Isaacs, W.A. (1971) Anaesthesia in patients with abnormal haemoglobin syndromes. A preliminary report. British Journal of Anaesthesia, 43, 1159. 7 Oduro, K.A. & Searle, J.F. (1972) Anaesthesia in sickle-cell states: a plea for simplicity. British Medical Journal, 4, 596. 8 Lehmann, H. (1973) Anaesthesia in sickle-cell states. British Medical Journal, 1, 290. Citing Literature Volume28, Issue3May 1973Pages 339-341 ReferencesRelatedInformation

Dynamics of glomerular ultrafiltration in the rat. II. Plasma-flow dependence of GFR.

Dynamics of glomerular ultrafiltration in the rat. II. Plasma-flow dependence of GFR.

A model of glomerular ultrafiltration in the rat.

A model of glomerular ultrafiltration in the rat.

Cardiovascular responses to various pulsatile pressures in the carotid sinus.

Cardiovascular responses to various pulsatile pressures in the carotid sinus.

Parenteral nutrition for the surgical patient. A review of indications and current practice.

Parenteral nutrition for the surgical patient. A review of indications and current practice.

Cardiovascular responses to hypertonic glucose. An aspect of resuscitation studied in patients with chronic renal failure.

Cardiovascular responses to hypertonic glucose. An aspect of resuscitation studied in patients with chronic renal failure.