Background: Fulminant type 1 diabetes mellitus (FT1DM) is characterized by rapid onset of disease and complete destruction of pancreatic β-cell function. It is associated with genetic susceptibility, viral infection, autoimmunity and pregnancy. In this report, the patient jumped from a 2-meter-high platform, which caused acute pancreatitis and then induced FT1DM. Clinical case: A 40-year-old male suffered from abdominal pain after jumping from a 2-meter-high platform on June 7, 2017, and the pain was relieved slightly after about half an hour. On June 8, abdominal pain was worsen with fever up to 38.5℃. He was treated with antibiotics in the local hospital. But abdominal pain gradually increased, and was relieved in prone position. He was hospitalized on June 10. Pancreatic CT showed pancreatic edema and obvious exudation. Amylase was 201U/L(15-115)and lipase normal. He was diagnosed as “acute pancreatitis” and treated with somatostatin, antibiotics, proton pump inhibitors and so on. Fasting blood glucose is 4.96mmol/L and body temperature is normal on June 11. On June 13, obvious thirst appeared. On June 14, laboratory examinations showed fasting blood glucose was 25.69mmol/L, HCO3- 13.24mmol/L, a white blood cell count of 20.22×109/L,neutrophil percentage 87.6%, amylase 872U/L, lipase 141U/L, and HbA1c was 5.69%. Insulin was given to reduce blood glucose. On June 16, fasting blood glucose was 16.49mmol/L. On June 26, somatostatin and proton pump inhibitors were stopped and abdominal pain disappeared. Hypoglycemic drugs were adjusted to insulin glargine and aspart. He was admitted to our hospital on July 1st. His height and weight were 170cm and 59.5kg, respectively (body mass index 20.95kg/m2). Serum glucose was very high (15.24mmol/L), while HbA1c was relatively low (6.8%). Fasting, normal postprandial 1h and 2h C-peptide levels were zero. Islet-related autoantibodies against GAD and ZnT8 were positive and others were negative. Pancreatic magnetic resonance imaging and MRCP were normal. IgG, IgG4, complement C3, C4, antinuclear antibody spectrum, vasculitis group, antibody to beta 2 glycoprotein, anticardiolipin antibodies, thyroid peroxidase antibody and thyroglobulin antibody were negative. He was discharged after blood glucose was controlled with insulin glargine and aspart. The patient had class II HLA gene showed DQB1*04:01-DRB1*04:05 haplotype. Conclusion: In the background of hereditary susceptibility, acute pancreatitis was induced by jumping off a 2-meter-high platform, and leaded to FT1DM. This is the first case of FT1DM caused by jumping down from a high place.