Compressor Stations Of Gas Pipelines Research Articles

The Experience of Gas Pipeline Stress-Strain State Control With Usage of the Metal Magnetic Memory Method as Compared With Conventional Methods and Stress Control Means

Test methods and means for characterising the stress-strain-state (SSS) of pipes in a gas pipeline compressor station were compared. At first the SSS determination methods and means were evaluated under laboratory conditions, and then under field conditions. The test objects were a special hydraulic test bench; supercharger manifold piping systems; a compressor station input loop; a duster manifold piping. For calibration artificial strains were impressed to the last two objects. The following SSS determination methods and means were applied: strain measurement; magnetic Barkhausen noise; metal magnetic memory (MMM) method; amplitude-phase frequency characteristics (APFC) method; hardness measurements; calculations; design-experiment method.

Gas collector of a new generation for compressor stations of transmission gas pipelines

Gas collector of a new generation for compressor stations of transmission gas pipelines

Evaluating the endurance of welded joints in manifold pipes of compressor stations of transmission gas pipelines

Evaluating the endurance of welded joints in manifold pipes of compressor stations of transmission gas pipelines

Prefabricated blocks of equipment for commercial-gas treatment plant for gas pipeline compressor stations

Prefabricated blocks of equipment for commercial-gas treatment plant for gas pipeline compressor stations

Triaryl phosphate poisoning in cattle.

Clinical signs, pathologic changes and biochemical changes occurred in cattle with natural and experimental triaryl phosphate poisoning. Natural poisoning was caused by triaryl phosphates escaping from a gas pipeline compressor station. The clinical signs were posterior motor paralysis, dyspnea, diarrhea and agalactia. Experimental doses of 1/2-1 gm/kg body weight of these organophosphate compounds caused depression of cholinesterase and axonal degeneration in the spinal cord.