Abstract
in the absence of obstructive CAD. Because the usual hemodynamic factors do not explain the occurrence of the intercoronary communications in these 2 cases, they probably represent large, low-resistance congenital communications. Since neither the previously described patient! nor our own had any electrocardiographic or scintigraphic evidence of myocardial ischemia, even during exercise, the intercoronary communications in these patients probably have little or no functional significance. 1. Baretdi 0. Coronary heart disease: significance of the anatomic lesions. Am Heart J 1973;65:1-5. 2. GaminI GG, Gruto Da Costa BC. The coronary collateral circulation in living man. Am J Cardiol 1969;24:393-400. 3. Levin DC. Pathways and functional significance of the coronary collateral circutation. Circulation 1974;50:831-637. 4. En9 C, Pattersen RE, Horowttz SF, Halgash DA, Plchard AD, Mldwall J, Herman MV, Gerlln R. Coronary collateral function during exercise. Circulation 1962;66:309-316. 5. Loll PM, Wessler S, Schlesinger MH. interarterial coronary anastomoses in the human heart, with particular reference to anemia and relative cardiac anoxia. Circulation 1951;4:797-615. 6. Wilner BH, Mills RM, Starobln 0, Llngley JF. lntracoronary anastomosis in the absence of obstructive lesions of the coronary arteries. Chest 1979; 76:466-469.
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