Abstract
Atrial natriuretic peptide (ANP) and brain natriuretic peptide (BNP) are synthesized and secreted from the ventricular myocardium after the onset of myocardial infarction (MI), and plasma concentrations of ANP and BNP are significantly correlated with hemodynamic variables and residual left ventricular (LV) function. 1‐4 Furthermore, plasma concentrations of these peptides may be used as simple biochemical predictors of prognosis after MI. 5,6 LV dilatation after MI may occur due to expansion of the infarct site, 7 hypertrophy of noninfarcted myocardium, or maladaptive LV “ remodeling.” 8,9 We hypothesized that plasma natriuretic peptide concentration correlated with LV remodeling. We studied the relation between plasma natriuretic peptide concentrations 1 month after the onset of MI and LV dilatation.
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