Abstract
ESULTS: Treatment of carcinoid cells with TTN resulted in proressive phosphorylation of GSK-3 beta, indicating a doseependent inhibition of the GSK-3 beta signaling pathway. Imporantly, TTN significantly inhibited pancreatic and pulmonary arcinoid cellular proliferation. Furthermore, inhibition of GSK-3 eta by TTN resulted in a dose-dependent reduction in CgA and SCL1, suggesting a progressive inhibition of bioactive hormone roduction.
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