Abstract
To the Editor: A single nucleotide polymorphism in Type 2 deodinase ( Dio2 ) gene (A/G) in humans, in which a threonine (Thr) changes to alanine (Ala) at codon 92 (Thr92Ala), has been associated with a lower glucose disposal rate and higher insulin resistance in type 2 diabetes (DM2) patients.1,2 Nevertheless, these findings were not replicated in larger studies.3,4 Recently, Gumieniak et al reported that the Ala allele increases the risk for development of arterial hypertension.5 Since insulin resistance can cause hypertension, we sought to extend …
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