Abstract
An acute respiratory syndrome caused by SARS-CoV2 was declared a pandemic by the World Health Organization. Current data in the world and in Brazil show that approximately 40% of patients who died have some type of cardiac comorbidity. There are also robust reports showing an increase in IL-6 / IL-1B / TNF-alpha and the presence of lymphopenia in patients with COVID-19. Our team and others have shown that increased cytokines are the link between arrhythmias/Left ventricular dysfunction and the [...]
Highlights
The Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) is the RNA virus responsible for the Coronavirus Disease 19 (COVID-19)
Cardiovascular compromise due to COVID-19 was associated with arrhythmias (16%), myocardial ischemia (10%), myocarditis (7.2%) and shock (1-2%).[3]
A number of cases of heart arrhythmias have been described in the course of severe COVID-19, but understanding the role played by cytokine storm and putative viral myocarditis or even direct tissue infection is complicated by the administration of several drugs that might alter cardiac rhythm, and by the effects of refractory hypoxemia on heart conduction system
Summary
The Role of the Immune System on the Cardiac Complications Observed in SARS-CoV-2. Larissa Carvalho Vigário,[1] Guilherme Muradas,[1] Claudia Neto Paiva,[3] Emiliano Medei[1,2,4].
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