Abstract
AbstractPre‐treatment with abscisic acid (ABA) can protect oats (Avena sativa) against damage from the herbicide diclofop‐methyl. Examination of the uptake and translocation of diclofop‐methyl showed that although 20 μg ABA induced varying degrees of stomatal closure, ABA had no significant effect on the amount of herbicide entering the leaf. Translocation of [14C] diclofop‐methyl was also unafected by ABA treatment. Electrolyte leakage studies demonstrated that ABA prevented the loss of plasmalemma integrity normally associated with diclofop‐methyl damage. It was concluded that ABA protection was by active metabolism, and an increase in the tolerance of the plasmalemma to diclofop‐methyl.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
