Abstract
Spermatogenic failure in men with non-obstructive azoospermia (NOA) involves mechanisms that are not well understood. This reflects the complexity of the gene signaling pathways required for normal spermatogenesis. Gene expression microarrays allow for an in-depth analysis of these pathways and a comprehensive assessment of altered cellular function. Thus, they aid in the discovery of new gene defects causing non-obstructive spermatogenesis.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
