Abstract
The notion of somatic mutation has found little favour among experimental biologists or clinicians. Burnet (1959(b)) has attributed this lack of appeal to the prevailing ethos: ‘It is almost an inescapable characteristic of those educated in Western and scientific habits of thought to believe that effects have a definable cause and that undesired phenomena can always be prevented or cured… It is diminishing (Burnet’s italics) to consider our helplessness in the face of the major limitations of life, the inevitable accident that sooner or later introduces a flaw into the copying of a genetic pattern’.
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