Abstract
Aneuploidy is the most common cause for spontaneous miscarriage and congenital disorders. Two major mechanisms have been identified as drivers of aneuploidy, the extended dictyate arrest after DNA replication and meiotic recombination, including the vulnerable bivalent recombination configurations between homologous chromosomes and cohesion with sister chromatids. This study examined the transcriptome of both aneuploid and haploid oocytes to assist in the identification of the molecular signature of meiotic errors to further our understanding of oocyte aneuploidy.
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