Abstract
Evidence is reviewed which indicates that CAEV persists in infected goats with variable and restricted expression. Immunosuppression prevents the lesions caused by CAEV. Arthritis is enhanced in CAEV-challenged goats that have been immunized with inactivated CAEV or are persistently infected with CAEV. A large part of the synovial fluid immunoglobulin is antibody against virion surface glycoproteins. These observations support the hypothesis that the immune response to viral antigens is a major determinant of CAEV arthritis.
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