Abstract
In-stent restenosis (ISR) and stent thrombosis are the main adverse reactions to coronary stent implantation in which an important role is played by individual susceptibility, along with procedural and stent related factors [1]. In particular, the individual inflammatory reaction plays an important role in vessel response to the implanted stent [1]. Indeed, inflammatory status, as assessed by C-reactive protein levels, is well known to predict the risk of ISR after bare-metal stent (BMS) implantation [2], but not after drug-eluting stent (DES) implantation [3], although it appears to predict the risk of DES thrombosis [4].
Published Version
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