Abstract
1. 1. Iron overload and iron depletion respectively potentiated and diminished the porphyrinogenic effect of hexachlorobenzene in rats, as assessed by hepatic, renal and colonie activity of uroporphyrinogen decarboxylase and by urinary and faecal porphyrin excretion. 2. 2. In the early phase of hexachlorobenzene intoxication the rat undergoes a definite porphyric phase different from the porphyria cutanea tarda-like phase which occurs later. 3. 3. Preliminary indications are that faecal and urinary uroporphyrin excretion can be correlated with colonie and renal uroporphyrinogen decarboxylase activity respectively.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
