Abstract
There is increasing interest in the literature regarding the clinical delineation of suprascapular nerve (SSN) injuries. While the underlying etiology of SSN neuropathy remains elusive, multiple compression, traction, and inflammatory pathophysiologic cascades have been previously considered. The latter are often readily grouped together with other clinical manifestations of SSN dysfunction and inevitably become labeled as idiopathic. The anatomically tortuous course of the SSN predisposes it to direct mechanical compression at the suprascapular and spinoglenoid notches. Moreover, anatomic variations along the nerve's course as well as occupational or athletic overuse conditions, especially in overhead athletes, can predispose the SSN varying degrees of dysfunction.
Published Version
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
