Abstract
We have shown 1 that relative inagglutinability of Bact. typhosum and of B. pestis-caviae could be induced by growing these organisms in sterile gum tragacanth abscesses in mice. Experiments by one of us (Ecker) demonstrated that relative inagglutinability of Bact. typhosum could also be induced by growth in 20% sterile normal human serum broth (6 transfers), and that the serum of the atypical typhoid patient described by Blankenhorn, Ecker and King2 produced a similar alteration of a normal strain to the same degree. The strain isolated in this case, however, remained inagglutinable under the same conditions and was not made more agglutinable by growth in convalescent typhoid serum, while the ordinary laboratory strain sedimented out in dilutions from 1: 80-5,000 in fine floccules following 6 transfers in this serum broth.
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