Abstract
Liver transplantation (TxH) is the only effective treatment for terminal liver disease, whether chronic, subacute or acute. The surgical procedure of liver transplantation, the largest current surgery in the human body contributes to the increase of catecholamine concentrations, favoring the occurrence of Takotsubo Syndrome (STK), which is a transient dysfunction of the left ventricle caused by an increase of catecholamines during events of high physiological stress. The importance of considering the diagnosis of STK in the hypothesis formulated for hemodynamic decompensation aims to avoid its complications, especially cardiogenic shock. The management of post-TxH patients with STK is not well established, but there are some strategies described in the literature. The knowledge of the underlying cause for the complications gives greater efficacy to the therapeutic measures to be chosen, providing good evolution to the TxH receptors with STK.
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