Abstract

Uterine fibroids represent the most common benign tumors in women in reproductive age. It has been reported that they arise from a single dysregulated myometrial smooth muscle cell. However, the underlying tumorigenic mechanism remains unclear and because of that, surgery has been the gold standard for their treatment. Our primary motivation stems from the need to better understand the cellular hierarchy of uterine fibroids and myometrium, leveraging the high resolution of single cell RNAseq. We aim to identify cell types and states that are unique to the fibroids, based on their molecular signatures.

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