Abstract
Thymic stromal lymphopoietin (TSLP) plays an important role in the pathogenesis of atopic dermatitis (AD) by promoting dendritic cell-mediated activation of the Th2 inflammatory response [1]. The lipophilic yeasts Malassezia globosa and Malassezia restricta are members of the cutaneous microflora and act as exacerbating factors in AD [2]. Despite the in vivo expression and potential importance of keratinocyte-derived TSLP in AD [1], no studies have tested the influence of the presence of Malassezia yeast cells on the production of TSLP by human keratinocytes.
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