Abstract
In experiment on white rats-males of Vistar line in weight of 200–250 g simulated a 30-minute ischemia/reperfusion of distal part of small intestine, investigated a condition of monooxygenase and reductase chains of microsome of hepatocytes endoplasmic reticulum. Decrease of all studied parameters microsomal oxidations in early terms investigation (72 hour), against the intensity growth of lipid peroxidation testifies about malfunction of detoxication liver functions. Preliminary abdominal introduction of glucoproteides a-globulin fraction of human serumand carnitine to modeling ischemia/reperfusion of the small intestine protect cytochromes P-450 and b5 from oxidative damage, support the active functioning of the electron transport chain, contributing to improved energy cells by reducing the rate of lipid peroxidation and changes in endogenous respiration rate.
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