Abstract
Background: Th17 cells play a key role in several autoimmune disorders including multiple sclerosis (MS) and inflammatory bowel disease (IBD). Their differentiation is mainly regulated by a concerted action of several nuclear receptors such as RORgammaT, RORalpha and, as suggested recently, NFIL3 and NR1D1 as part of the peripheral clock. However, little is known about possibly disease-specific contributions of these elements of Th17 biology.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
