Abstract

Review of: "Aspirin-triggered resolvin D1 reduces parasitic cardiac load by decreasing inflammation through N-formyl peptide receptor 2 in a chronic murine model of Chagas disease"

Highlights

  • The research article entitled "Aspirin-triggered resolvin D1 reduces parasitic cardiac load by decreasing inflammation through N-formyl peptide receptor 2 in a chronic murine model of Chagas disease" by Ileana Carrillo et al, investigates the role of Aspirin-triggered resolvin D1 (AT-RvD1) as a pro-resolving mediator of inflammation that acts through N-formyl peptide receptor 2 (FPR2) using a FPR2 knockout murine model of chronic Trypanosoma cruzi infection

  • The data provided in the manuscript is important and interesting in connecting a link between AT-RvD1 as an attractive therapeutic activity due to its regulatory effect on the inflammatory response at the pathophysiology to cardiac pathology during T. cruzi infection

  • The manuscript is an original research article and I recommend it, it suffers with the following minor concerns: Materials and methods section 1

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Summary

Introduction

Review of: "Aspirin-triggered resolvin D1 reduces parasitic cardiac load by decreasing inflammation through N-formyl peptide receptor 2 in a chronic murine model of Chagas disease" Potential competing interests: The author(s) declared that no potential competing interests exist.

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