Abstract
Review of: "Aspirin-triggered resolvin D1 reduces parasitic cardiac load by decreasing inflammation through N-formyl peptide receptor 2 in a chronic murine model of Chagas disease"
Highlights
The research article entitled "Aspirin-triggered resolvin D1 reduces parasitic cardiac load by decreasing inflammation through N-formyl peptide receptor 2 in a chronic murine model of Chagas disease" by Ileana Carrillo et al, investigates the role of Aspirin-triggered resolvin D1 (AT-RvD1) as a pro-resolving mediator of inflammation that acts through N-formyl peptide receptor 2 (FPR2) using a FPR2 knockout murine model of chronic Trypanosoma cruzi infection
The data provided in the manuscript is important and interesting in connecting a link between AT-RvD1 as an attractive therapeutic activity due to its regulatory effect on the inflammatory response at the pathophysiology to cardiac pathology during T. cruzi infection
The manuscript is an original research article and I recommend it, it suffers with the following minor concerns: Materials and methods section 1
Summary
Review of: "Aspirin-triggered resolvin D1 reduces parasitic cardiac load by decreasing inflammation through N-formyl peptide receptor 2 in a chronic murine model of Chagas disease" Potential competing interests: The author(s) declared that no potential competing interests exist.
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