Abstract
In their interesting and novel study in this issue of The Lancet Neurology, Lindley and colleagues1 report microvascular abnormalities in the retinal vessels of patients who have had ischaemic small vessel stroke (lacunar stroke). These retinal changes, which include a smaller arteriole:venule ratio, arteriovenous crossing abnormalities, enhanced arteriolar light reflex, and wider retinal venules in patients without diabetes, support an underlying mechanism of intracerebral vessel narrowing in lacunar infarction that has a non-atherothrombotic origin.
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