Abstract
Heart failure patients and heart transplant (HT) recipients (HTRs) have impaired cognitive function, secondary to reduced cerebral blood flow (CBF).1,2 Currently, only 3 studies have measured CBF in HTRs.1–3 Gruhn et al1 reported that CBF was 30% lower in heart failure patients and normalized 1 month after HT. They speculated that lower post-surgical hematocrit levels reduced the viscosity in the blood and that this could account for the increase in CBF. Massaro et al2 demonstrated a CBF increase of ∼53% at 1 month after HT; however, they were unable to correlate the increase to lower hematocrit levels.
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