Abstract
Antioxidants & Redox SignalingVol. 9, No. 7 Forum EditorialRedox Imbalance in DiabetesMelvin R. Hayden and James R. SowersMelvin R. HaydenSearch for more papers by this author and James R. SowersSearch for more papers by this authorPublished Online:17 May 2007https://doi.org/10.1089/ars.2007.1640AboutSectionsPDF/EPUB ToolsPermissionsDownload CitationsTrack CitationsAdd to favorites Back To Publication ShareShare onFacebookTwitterLinked InRedditEmail FiguresReferencesRelatedDetailsCited ByPersistence of improved glucose homeostasis in Gclm null mice with age and cadmium treatmentRedox Biology, Vol. 49Mitochondrial Regulation of Diabetic Kidney Disease27 September 2021 | Frontiers in Medicine, Vol. 8The Link between Type 2 Diabetes Mellitus and the Polymorphisms of Glutathione-Metabolizing Genes Suggests a New Hypothesis Explaining Disease Initiation and Progression28 August 2021 | Life, Vol. 11, No. 9Immunometabolic bases of type 2 diabetes in the severity of COVID-19World Journal of Diabetes, Vol. 12, No. 7NADH/NAD+ Redox Imbalance and Diabetic Kidney Disease14 May 2021 | Biomolecules, Vol. 11, No. 5Disruption of mitochondrial redox homeostasis by enzymatic activation of a trialkylphosphine probe1 January 2021 | Organic & Biomolecular Chemistry, Vol. 19, No. 12An Immediate and Long-Term Complication of COVID-19 May Be Type 2 Diabetes Mellitus: The Central Role of β-Cell Dysfunction, Apoptosis and Exploration of Possible Mechanisms13 November 2020 | Cells, Vol. 9, No. 11Type 2 Diabetes Mellitus Increases The Risk of Late-Onset Alzheimer’s Disease: Ultrastructural Remodeling of the Neurovascular Unit and Diabetic Gliopathy29 September 2019 | Brain Sciences, Vol. 9, No. 10Monitoring Dynamic Cellular Redox Homeostasis Using Fluorescence-Switchable Graphene Quantum Dots1 December 2016 | ACS Nano, Vol. 10, No. 12Store-operated calcium entry and diabetic complications14 October 2015 | Experimental Biology and Medicine, Vol. 241, No. 4Nuclear Factor κB Mediates Suppression of Canonical Transient Receptor Potential 6 Expression by Reactive Oxygen Species and Protein Kinase C in Kidney CellsJournal of Biological Chemistry, Vol. 288, No. 18Molecular and metabolic mechanisms of cardiac dysfunction in diabetesLife Sciences, Vol. 92, No. 11La variabilité glycémique en réanimationAnnales Françaises d'Anesthésie et de Réanimation, Vol. 31, No. 12The emerging role of cardiovascular risk factor-induced mitochondrial dysfunction in atherogenesis9 December 2009 | Journal of Biomedical Science, Vol. 16, No. 1QUANTITATIVE REDOX SCANNING OF TISSUE SAMPLES USING A CALIBRATION PROCEDURE21 November 2011 | Journal of Innovative Optical Health Sciences, Vol. 02, No. 04Attenuation of Endocrine-Exocrine Pancreatic Communication in Type 2 Diabetes: Pancreatic Extracellular Matrix Ultrastructural AbnormalitiesJournal of the CardioMetabolic Syndrome, Vol. 3, No. 4Ultrastructure of Islet Microcirculation, Pericytes and the Islet Exocrine Interface in the HIP Rat Model of Diabetes1 September 2008 | Experimental Biology and Medicine, Vol. 233, No. 9Treating hypertension while protecting the vulnerable islet in the cardiometabolic syndromeJournal of the American Society of Hypertension, Vol. 2, No. 4 Volume 9Issue 7Jul 2007 InformationCopyright 2007, Mary Ann Liebert, Inc.To cite this article:Melvin R. Hayden and James R. Sowers.Redox Imbalance in Diabetes.Antioxidants & Redox Signaling.Jul 2007.865-867.http://doi.org/10.1089/ars.2007.1640Published in Volume: 9 Issue 7: May 17, 2007PDF download
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