Abstract
RECURRENT inhibition via interneurones (Renshaw cells) was recently invoked by Brooks and Wilson1 to account for changes in monosynaptic spinal reflexes following antidromic activation. This mechanism, which confines stretch reflexes to their paths of afferent origin, operates through a cholinergic synapse between the motoneurone axon collateral and the Renshaw cell2, which liberates an unknown inhibitory transmitter. Presumably the same mechanism functions with respect to disynaptic pathways through the cord. The purpose of this communication is to describe an analogous system in the cerebral cortex of dogs, operated under ether and immobilized by succinylcholine.
Published Version
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
