Abstract
Renal involvement by tuberculosis leads to fibrosis and multi focal stricturing of calyces with formation of hydrocalyces. The obstructed calyceal system accumulates caseous pus within it which eventually calcifies. This obstructed and calcified kidney is non-functional and is referred to ‘putty kidney’ and this cascade of fibrosis, stricture formation, calcification with complete loss of function is called as autonephrectomy (1,2).
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