Abstract
The proposal that the hepatic fat accumulation induced by ingestion of carbon tetrachloride by rats is due to increased fatty acid mobilization has been tested. The data show that neither the plasma volume as determined following I 131 injection of serum albumin nor the free fatty acid concentration was changed by carbon tetrachloride treatment. Since the disappearance of injected palmitate 1-C 14 was not increased following ingestion of this hepatotoxin, it appears unlikely that increased fatty acid mobilization plays a role in the initial development of a fatty liver.
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