Abstract
Preeclampsia is a hypertensive disorder of pregnancy that can present with proteinuria or end-organ damage. Preeclampsia occurs in approximately 5% of pregnancies and is associated with significant fetal and maternal morbidity that can extend beyond the period of pregnancy. The exact pathogenesis of preeclampsia is still unclear; however, evidence supports a role for placental ischemia and disordered development and remodeling of the placental vasculature.1 These processes are especially relevant in “placental” or “early” preeclampsia, in which they are thought to contribute to an altered repertoire of placenta-derived factors that promote maternal endothelial dysfunction.
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