Abstract
Antibody-mediated rejection (AMR) is emerging as an important cause of solid organ transplantation failure. The role of AMR in lung allografts is a source of ongoing investigation, as well as the identification of more sensitive and accurate diagnostic features. The mammalian target of rapamycin (mTOR) pathway, in particular phosphorylated P70 S6 kinase (S6K) and S6 ribosomal protein (S6RP), is reported to be associated with AMR in heart and kidney transplantation. The main goal of the study was to evaluate S6K and S6RP expression in lung transplanted (LT) patients with AMR from different LT centers.
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