Abstract
Nitric oxide (NO) plays a role on parasympathetic reactivation after a maximal exercise test. Previous studies have shown that endothelial nitric oxide synthase (NOS3) gene polymorphisms reduce NO bioavailability. Therefore subjects with NOS3 polymorphisms may have lower parasympathetic reactivation after exercise. The aim of this study was to investigate the influence of three NOS3 gene polymorphisms (−786T>C, intron 4b4a and 894G>T) on parasympathetic reactivation within the first 5min after a maximal cardiopulmonary exercise test.
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