Abstract
Full-thickness articular cartilage defects of the knee are a significant cause of patient morbidity. As treatment options have expanded, existing treatment algorithms have become increasingly complex (1,2). Osteochondral allograft and autograft transplantation (in this article, OAT refers exclusively to osteochondral allograft transplantation) have received increasing attention for the treatment of full-thickness cartilage defects since their initial study (3-5).
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